Genetic alterations promote childhood obesity

A revealing fact about the causes of childhood obesity is that it could be related to a genetic factor. That is, suffering from it would not only be linked to bad eating habits, but also to an alteration based on the loss of fragments in the DNA.

British researchers at the University of Cambridge found that, apparently, the absence of a gene on chromosome 16, which the brain needs to respond to the leptin hormone that controls appetite, would cause the problem.

Children with this failure on chromosome 16 "feel very hungry," say the study authors, which could give us a clue about the great eating needs of obese children.

That childhood obesity could have a specific genetic cause opens a new landscape in the scientific world. On the one hand, an attenuator is thought for parents accused of negligence in the face of their children's obesity, and on the other, that knowing more information about the medical causes of the disease, new solutions can emerge.

Anyway, this would not mean that bad eating habits do not influence to trigger the disease, as well as other factors such as sedentary lifestyle, few hours of sleep or lack of exercise. But at least in certain genetic variations would be the answer to why a child becomes obese when he eats as badly as one who is not. Because it suffers from a certain chromosomal alteration that promotes childhood obesity.